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Journal Article

Citation

Takahashi M, Maemura K, Sawada Y, Yoshioka T, Sugimoto T. J. Trauma 1980; 20(11): 951-955.

Copyright

(Copyright © 1980, Lippincott Williams and Wilkins)

DOI

unavailable

PMID

6159479

Abstract

We found that in traumatic shock patients without pancreatic injury, hyperamylasemia occurs in high frequency [49 of 61 (80%)]. Isoenzyme studies of 19 of these patients revealed 18 of them (94%) to have the salivary type of hyperamylasemia. Further, based on the results of the present study, the salivary gland as the organ of origin and the permeability of its cell membranes are suggested as the mechanism for traumatic hyperamylasemia.


Language: en

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